Bacterial Invasiveness by R. R. Isberg (auth.), Professor Virginia L. Miller Ph.D.

By R. R. Isberg (auth.), Professor Virginia L. Miller Ph.D. (eds.)

Intracellular pathogens are accountable for a few very important ailments around the globe, together with tuberculosis, plague and bacillary dysentery. This quantity focusses on these intracellular pathogens which were studied so much commonly on the molecular, genetic, and mobile point. The reports try and combine the knowledge derived from those assorted techniques right into a cohesive photo. lately the access steps were defined on the molecular and genetic point, and the $64000 sign transduction occasions are being elucidated. it truly is now changing into transparent that there are either similarities and modifications either by way of the stairs concerned and of the genetic foundation of bacterial invasiveness. those reports of the "state of the artwork" offer a beginning from which to proceed.

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Cell-to-Cell Spread . . . . . . . . . . . . . . . . . . . . . . . . . . . 2 Tissue Invasion ........................................................ Portal of Entry ....................................... . . . . . . . . . Invasion and Inflammation ............................................... 34 34 35 5 Conclusion .......................................................... 35 References ................................................................ 37 1 Introduction Bacillary dysentery, or shigellosis, is caused by penetration of Shigella spp into the intestinal mucosa of the colon, where degeneration of the epithelium and a strong inflammatory reaction indicate sites of infection (LABREe et al.

Recent immunohistochemical analysis of rectal biopsies taken from patients with shigellosis has shown, in tissue samples characterized by severe inflammation, a predominance of such IL-l-producing cells as mononuclear cells, PM N leukocytes, and endothelial cells (RAQUIB et al. 1995). Invasion of lymphoid follicles is likely to be the trigger of this inflammatory process, which then subverts the epithelial architecture, thereby causing extensive invasion of the intestine. 5 Conclusion Invasion of a cell monolayer by Shigella involves numerous interactions between the bacterium and the host cell (Fig.

2 Intra- and Intercellular Dissemination ....................................... Biology of Intracellular Movement ......................................... The les Movement ..................................................... The Olm Movement .................................................... Genetics of Intercellular Dissemination ..................................... Intracellular Movement . . . . . . . . . . . . . . . . . . . . . . . . .

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